Infarct Size

Tissue Vitamin A Insufficiency Results in Adverse Ventricular Remodeling after Experimental Myocardial Infarction

Vitamin A / Left Ventricular Assist Device / Animals / Male / Heart rate / Medical Physiology / Rats / Myocardial Infarction / Tumor necrosis factor-alpha / Vitamin A deficiency / Body Weight / Wistar Rats / Infarct Size / Ventricular Remodeling / Diastolic Dysfunction/CHF / Matrix Metalloproteinase / Interferon gamma / Medical Physiology / Rats / Myocardial Infarction / Tumor necrosis factor-alpha / Vitamin A deficiency / Body Weight / Wistar Rats / Infarct Size / Ventricular Remodeling / Diastolic Dysfunction/CHF / Matrix Metalloproteinase / Interferon gamma

Modulación del sistema betaadrenérgico durante el infarto agudo de miocardio: justificación para un nuevo ensayo clínico

Research Design / Clinical Trial / Magnetic Resonance Imaging / Treatment Outcome / Humans / Left Ventricular Assist Device / Acute Myocardial Infarction / Myocardial Infarction / Clinical Study / Infarct Size / Reperfusion injury / Retrospective Study / Beta Blockers / Coronary artery / Left Ventricular Assist Device / Acute Myocardial Infarction / Myocardial Infarction / Clinical Study / Infarct Size / Reperfusion injury / Retrospective Study / Beta Blockers / Coronary artery

Modulación del sistema betaadrenérgico durante el infarto agudo de miocardio: justificación para un nuevo ensayo clínico

Research Design / Clinical Trial / Magnetic Resonance Imaging / Treatment Outcome / Humans / Left Ventricular Assist Device / Acute Myocardial Infarction / Myocardial Infarction / Clinical Study / Infarct Size / Reperfusion injury / Retrospective Study / Beta Blockers / Coronary artery / Left Ventricular Assist Device / Acute Myocardial Infarction / Myocardial Infarction / Clinical Study / Infarct Size / Reperfusion injury / Retrospective Study / Beta Blockers / Coronary artery

A novel peroxynitrite decomposer catalyst (FP-15) reduces myocardial infarct size in an in vivo peroxynitrite decomposer and acute ischemia-reperfusion in pigs

Nitric oxide / Female / Animals / Male / Nitrogen / Clinical Sciences / Myocardial Infarction / Swine / Coronary Circulation / Infarct Size / Coronary Artery Disease / Reperfusion injury / Ischemia–reperfusion / Superoxide Anion / Control Group / Reactive oxygen and nitrogen species / Clinical Sciences / Myocardial Infarction / Swine / Coronary Circulation / Infarct Size / Coronary Artery Disease / Reperfusion injury / Ischemia–reperfusion / Superoxide Anion / Control Group / Reactive oxygen and nitrogen species

Hypoxic postconditioning reduces cardiomyocyte loss by inhibiting ROS generation and intracellular Ca2+ overload

Physiology / Calcium / Anoxia / Reactive Oxygen Species / Animals / American / Medical Physiology / Superoxide Dismutase / Hydrogen Peroxide / Rats / Wistar Rats / Indexation / Primary Culture / Infarct Size / Cell communication / Cell Survival / Malondialdehyde / Superoxides / Intracellular Calcium / American / Medical Physiology / Superoxide Dismutase / Hydrogen Peroxide / Rats / Wistar Rats / Indexation / Primary Culture / Infarct Size / Cell communication / Cell Survival / Malondialdehyde / Superoxides / Intracellular Calcium

Reperfusion induces myocardial apoptotic cell death

Cardiovascular / Immunohistochemistry / Apoptosis / Western blotting / Dogs / Female / Animals / Male / Cell Death / Heart / Neutrophils / Western blot / Coronary Blood Flow / Gel electrophoresis / Infarct Size / Myeloperoxidase / Female / Animals / Male / Cell Death / Heart / Neutrophils / Western blot / Coronary Blood Flow / Gel electrophoresis / Infarct Size / Myeloperoxidase

Transgenic MMP-2 expression induces latent cardiac mitochondrial dysfunction

Mice / Animals / Biochemical / Creatine Kinase / Lipid peroxidation / Mitochondrial dysfunction / Myocardial Infarction / Myocardium / Necrosis / Infarct Size / Ischemia–reperfusion / Transgene Expression / Ischemic Preconditioning / Biochemistry and cell biology / Matrix Metalloproteinase / Mitochondrial dysfunction / Myocardial Infarction / Myocardium / Necrosis / Infarct Size / Ischemia–reperfusion / Transgene Expression / Ischemic Preconditioning / Biochemistry and cell biology / Matrix Metalloproteinase

Preconditioning delays Ca 2+ -induced mitochondrial permeability transition

Cardiology / Electron Microscopy / Cardiovascular / Calcium / Mitochondria / Apoptosis / Transition / Permeability / Animals / Cell / Male / Cyclosporine / Apoptose / Myocardial Infarction / Necrosis / Mitochondrial Permeability Transition Pore / Rabbits / Infarct Size / Preconditioning / Ischemic Preconditioning / Random Allocation / Immunosuppressive Agents / Apoptosis / Transition / Permeability / Animals / Cell / Male / Cyclosporine / Apoptose / Myocardial Infarction / Necrosis / Mitochondrial Permeability Transition Pore / Rabbits / Infarct Size / Preconditioning / Ischemic Preconditioning / Random Allocation / Immunosuppressive Agents

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